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The possibility of providing acetyl groups makes ALCAR able to maintain the intramitochondrial pathways, to reactivate coenzyme A, to reduce peroxidation and intracellular malonyl aldehyde levels, to act as a scavenger and to contribute to neurotransmitter synthesis due to the structural affinity to acetylcholine [17]

ALC operates via several mechanisms, inducing regeneration of injured nerve fibers, reducing oxidative stress, promoting DNA synthesis in mitochondria, and increasing NGF concentrations in neurons, thus promoting neurite extension

You may immediately notice an energy boost, ultimately increasing your metabolism

Brain-derived neurotrophic factor (BDNF) in traumatic brain injuryrelated mortality: interrelationships between genetics and acute systemic and central nervous system BDNF profiles